Author

B. E. Gewurz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

B. Mitra· 1 shared E. Burton· 1 shared E. M. Burton· 1 shared J. Voyer· 1 shared L. A. Murray-Nerger· 1 shared M. P. Weekes· 1 shared N. R. Beri· 1 shared R. Guo· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 12 mutual co-author links

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1 shared paperCancer Biology

B. Mitra

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MicrobiologyProceedings of the National Academy of SciencesPeer-reviewedJan 23, 2025

Epstein-Barr Virus BALF0/1 Subverts the Caveolin and ERAD Pathways to Target B-cell Receptor Complexes for Degradation

S. P. T. Yiu, M. P. Weekes, B. E. Gewurz

This paper identifies that EBV's BALF0/1 protein degrades B-cell receptor complexes by subverting caveolin and ERAD pathways, enhancing viral secretion.

DOI 10.1073/pnas.2400167122

humanproteomics

𝕏 PostBluesky

Cancer BiologybioRxivApr 10, 2023

Characterization of Target Gene Regulation by the Two Epstein-Barr Virus Oncogene LMP1 Domains Essential for B-cell Transformation

B. Mitra, N. R. Beri, R. Guo…·B. E. Gewurz

This study identifies TES1 as critical for LCL survival and characterizes its gene regulatory effects compared to TES2.

DOI 10.1128/mbio.02338-23

humanbulk RNA-seq

𝕏 PostBluesky

MicrobiologybioRxivFeb 21, 2022

Epstein-Barr virus latency programs dynamically sensitize B-cells to ferroptosis

E. M. Burton, J. Voyer, B. E. Gewurz

This study shows EBV-infected B-cells are sensitive to ferroptosis early in infection but become resistant later due to latency programs and lipid metabolism changes.

DOI 10.1073/pnas.2118300119

humanbulk RNA-seq

𝕏 PostBluesky