Author

B. Perkins

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Research Focus:cell signaling & trafficking· 2 Organism:zebrafish· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1

Frequent Co-Authors

A. Ibarra· 1 shared A. R. Moliterno· 1 shared A. R. Syage· 1 shared B. Anand-Apte· 1 shared B. Paun· 1 shared C. Bonifant· 1 shared C. Esteb· 1 shared D. Tantin· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperImmunology

A. Ibarra

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

OCA-B/Pou2af1 is sufficient to promote CD4+ T cell memory and prospectively identifies memory precursors

W. Sun, E. P. Hughes, H. Kim, J. Perovanovic, K. R. Charley, B. Perkins, J. Du, A. Ibarra, A. R. Syage, J. S. Hale, M. A. Williams, D. Tantin

This study shows that OCA-B expression promotes CD4+ T cell memory and identifies memory precursor cells using a reporter mouse line.

Proceedings of the National Academy of Sciences · Feb 27, 2024Peer-reviewedImmunology

The role of the atypical chemokine receptor CCRL2 in myelodysplastic syndrome and secondary acute myeloid leukemia

T. KARANTANOS, P. Teodorescu, B. Perkins, I. Christodolou, C. Esteb, R. Varadhan, E. Helmenstine, T. Rajkhowa, B. Paun, C. Bonifant, W. B. Dalton, L. P. Gondek, A. R. Moliterno, M. J. Levis, G. Ghiaur, R. J. Jones

This study identifies CCRL2 as a mediator of MDS and secondary acute myeloid leukemia growth through JAK2/STAT signaling, with potential therapeutic implications.

bioRxiv · Jul 29, 2021Cancer Biology

Primary cilia are present on endothelial cells of the hyaloid vasculature but are not required for the development of the blood-retinal barrier

L. M. Pollock, B. Perkins, B. Anand-Apte

The paper investigates whether endothelial cilia are required for blood-retinal barrier development in zebrafish, finding that cilia are present but not necessary for barrier integrity.

bioRxiv · Nov 5, 2019Cell Biology

Ciliary Genes arl13b, ahi1 and cc2d2a Differentially Modify Expression of Visual Acuity Phenotypes but do not Enhance Retinal Degeneration due to Mutation of cep290 in Zebrafish

B. Perkins, E. Lessieur, P. Song, G. Nivar, E. Piccillo, J. Fogerty, R. Rozic

This study examines how mutations in ciliary genes arl13b, ahi1, and cc2d2a modify visual acuity phenotypes in zebrafish with cep290 mutations, showing no enhancement of retinal degeneration but altered visual performance.

bioRxiv · Mar 6, 2019Cell Biology