Author

B. Sow

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. E. Carpenter· 1 shared A. Hajjar· 1 shared A. Lee· 1 shared A. Ng· 1 shared A. Skepner· 1 shared A. Tan· 1 shared A. Tavakoli· 1 shared C. Scheepers· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperBioengineering

A. E. Carpenter

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2 papers

Recent·Most cited

TDP43 aggregation at ER-exit sites impairs ER-to-Golgi transport

H. Wu, L. C. Wang, B. Sow, D. Leow, J. Zhu, K. Gallo, K. Wilsbach, R. Gupta, L. Ostrow, C. Yeo, R. Sobota, R. Li

The paper identifies TDP43 aggregation at ER-exit sites as a cause of impaired ER-to-Golgi transport in ALS.

bioRxiv · Jan 24, 2024Cell Biology

nELISA: A high-throughput, high-plex platform enables quantitative profiling of the inflammatory secretome

M. Dagher, G. Ongo, N. Robichaud, J. Kong, W. Rho, I. Teahulos, A. Tavakoli, S. Bovaird, S. Merjaneh, A. Tan, K. Edwardson, C. Scheepers, A. Ng, A. Hajjar, B. Sow, , D. Juncker

nELISA enables high-throughput, high-plex protein profiling of the inflammatory secretome, revealing 447 significant cytokine responses across multiple donors and conditions.

bioRxiv · Apr 17, 2023 · PreprintBioengineering