C. Favard

HIV-1 diverts actin debranching mechanismsfor particle assembly and release in CD4 Tlymphocytes.

This study shows HIV-1 uses actin debranching to enhance virus assembly and release by recruiting Arpin in CD4 T lymphocytes.

Quantification of membrane binding and diffusion using Fluorescence Correlation Spectroscopy diffusion laws

This study develops an FCS diffusion law to quantify membrane binding and diffusion of proteins, validated with simulations and experiments on retroviral Gag proteins.

Optimized production and fluorescent labelling of SARS-CoV-2 Virus-Like-Particles to study virus assembly and entry.

This paper optimizes the production of SARS-CoV-2 Virus-Like Particles (VLPs) and demonstrates their fluorescent labeling for studying virus assembly and entry mechanisms.

Full assembly of HIV-1 particles requires assistance of the membrane curvature factor IRSp53.

This study shows that IRSp53 assists HIV-1 in forming membrane curvature necessary for complete particle assembly.

HIV-1 Gag specifically restricts PI(4,5)P2 and cholesterol mobility in living cells creating a nanodomain platform for virus assembly.

Stick-slip dynamics of cell adhesion triggers spontaneous symmetry breaking and directional migration

Cells break symmetry through mechanical instabilities causing stochastic adhesion detachment, initiating migration without pre-established polarity.

Self assembly of HIV-1 Gag protein on lipid membranes generates PI(4,5)P2/Cholesterolnanoclusters.

This study shows HIV-1 Gag self-assembly creates PIP2/cholesterol nanoclusters on lipid membranes during virus assembly.