Author

C. Graffmann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

A. Baranowsky· 1 shared A. Donat· 1 shared A. Ivanov· 1 shared A. Rahmani· 1 shared C. Erdmann· 1 shared D. Beule· 1 shared D. Jahn· 1 shared E. Otto· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Baranowsky

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Increased beta2-adrenergic signaling is a targetable stimulus essential for bone healing by promoting callus neovascularization

D. Jahn, P. R. Knapstein, E. Otto, P. Köhli, J. Sevecke, F. Graef, C. Graffmann, M. Fuchs, S. Jiang, M. Rickert, C. Erdmann, J. Appelt, L. Revend, Q. Küttner, J. Witte, , J. Keller

This study identifies beta2-adrenergic signaling as a key driver of bone healing by enhancing callus neovascularization, with implications for therapeutic strategies and the risks of beta-blockers.

bioRxiv · Jul 14, 2023Cell Biology