Author

C. J. Langouët-Astrie

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:metagenomics· 1

Frequent Co-Authors

A. R. Horswill· 3 shared E. P. Schmidt· 3 shared A. L. Haeberle· 1 shared B. A. Duerkop· 1 shared B. D. McCollister· 1 shared C. L. Erzen· 1 shared C. Robertson· 1 shared D. Frank· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersMicrobiology

A. R. Horswill

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MicrobiologybioRxivJan 10, 2023

Aging-associated augmentation of gut microbiome virulence capability drives sepsis severity.

J. F. Colbert, J. M. Kirsch, C. L. Erzen…·E. P. Schmidt

This study shows that aging increases gut microbial virulence, contributing to more severe sepsis in older adults.

DOI 10.1128/mbio.00052-23

mousemetagenomics

𝕏 PostBluesky

MicrobiologybioRxivPreprintDec 13, 2022

Host-derived protease promotes aggregation of Staphylococcus aureus by cleaving the surface protein SasG

H. Crosby, K. C. Keim, J. Kwiecinski…·A. R. Horswill

This study shows that host-derived protease cleaves SasG, promoting S. aureus aggregation and virulence in mouse infections.

DOI 10.1101/2022.12.13.520364

mouseproteomics

𝕏 PostBluesky

MicrobiologybioRxivJun 27, 2022

Group B Streptococcus Adaptation Promotes Survival in a Hyper-inflammatory Diabetic Wound Environment

R. A. Keogh, A. L. Haeberle, C. J. Langouët-Astrie…·K. S. Doran

This study shows that Group B Streptococcus adapts to the hyper-inflammatory environment of diabetic wounds by upregulating virulence factors, leading to increased survival.

DOI 10.1126/sciadv.add3221

mousebulk RNA-seq

𝕏 PostBluesky