Author

C. S. Olie

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Damianou· 2 shared A. Pinto-Fernandez· 2 shared B. M. Kessler· 2 shared E. van der Wal· 2 shared J. C. de Greef· 2 shared V. Raz· 2 shared A. Chiarenza· 1 shared B. den Hamer· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

A. Damianou

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cell BiologybioRxivPreprintApr 1, 2022

USP18 is an essential regulator of muscle cell differentiation and maturation

C. S. Olie, A. Pinto-Fernandez, A. Damianou…·B. M. Kessler

This study identifies USP18 as a critical regulator of muscle cell differentiation and maturation, independent of its role in the ISG response.

DOI 10.1101/2022.04.01.486741

mouseexperimental assay

𝕏 PostBluesky

Cancer BiologybioRxivPreprintMar 31, 2020

Deep analysis of the USP18-dependent ISGylome and proteome unveils important roles for USP18 in tumour cell antigenicity and radiosensitivity

A. Pinto-Fernandez, M. Salio, T. Partridge…·B. M. Kessler

This study identifies novel ISGylation targets regulated by USP18, showing its role in enhancing tumour cell antigenicity and reducing radiosensitivity in CML cells.

DOI 10.1101/2020.03.31.005629

humanproteomics

𝕏 PostBluesky

Cell BiologybioRxivPreprintJan 15, 2020

Reduced PABPN1 levels causes cytoskeleton disorganization and aberrant differentiation

V. Raz, E. van der Wal, C. Domagoj…·B. Kessler

This study shows that reduced PABPN1 levels disrupt cytoskeletal organization and impair muscle differentiation, with actin overexpression improving these defects.

DOI 10.1101/2020.01.15.907311

mouseproteomics

𝕏 PostBluesky