Author

D. A. Hoagland

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

B. tenOever· 2 shared R. Moeller· 2 shared A. C. Borczuk· 1 shared A. Kodra· 1 shared A. Lachmann· 1 shared A. Ma'ayan· 1 shared A. Murphy· 1 shared A. Ruiz· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersMicrobiology

B. tenOever

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MicrobiologybioRxivJan 18, 2022

SARS-CoV-2 infection results in lasting and systemic perturbations post recovery

J. J. Frere, R. A. Serafini, K. D. Pryce…·B. tenOever

This study compares short- and long-term systemic responses to SARS-CoV-2 and IAV in golden hamsters, finding persistent inflammation in olfactory tissues post-recovery.

DOI 10.1126/scitranslmed.abq3059

mousebulk RNA-seq

𝕏 PostBluesky

Cell BiologybioRxivSep 20, 2020

Common genetic variation in humans impacts in vitro susceptibility to SARS-CoV-2 infection

K. Dobrindt, D. A. Hoagland, C. Seah…·K. J. Brennand

This study shows that a common genetic variant in the FURIN gene affects SARS-CoV-2 infection in vitro, suggesting genetic factors contribute to clinical variability in COVID-19.

DOI 10.1016/j.stemcr.2021.02.010

humanexperimental assay

𝕏 PostBluesky

MicrobiologybioRxivPreprintJul 12, 2020

Modulating the transcriptional landscape of SARS-CoV-2 as an effective method for developing antiviral compounds

D. A. Hoagland, D. J. B. Clarke, R. Moeller…·B. R. tenOever

This study identifies antiviral compounds that reduce SARS-CoV-2 viral load by counteracting the virus-induced transcriptional landscape, without affecting viral entry or host antiviral response.

DOI 10.1101/2020.07.12.199687

humanbulk RNA-seq

𝕏 PostBluesky