Author

D. A. Stevens

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Animal Behavior & Cognition· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

A. Bhat· 1 shared A. L. Arthur· 1 shared A. P. Ryan· 1 shared B. Cao· 1 shared B. V. Tsu· 1 shared C. Beierschmitt· 1 shared C. Patterson· 1 shared C. Rubel· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCell Biology

A. Bhat

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Antiviral function and viral antagonism of the rapidly evolving dynein activating adapter NINL

D. A. Stevens, C. Beierschmitt, S. Mahesula, M. R. Corley, J. Salogiannis, B. V. Tsu, B. Cao, A. P. Ryan, S. L. Reck-Peterson, M. D. Daugherty

This study identifies NINL as an antiviral innate immune component targeted by viruses through proteolytic cleavage, revealing its role in interferon response and viral antagonism.

bioRxiv · Jul 11, 2022Microbiology

Evolutionarily related small viral fusogens hijack distinct but modular actin nucleation pathways to drive cell-cell fusion

K. M. C. Chan, A. L. Arthur, J. Morstein, M. Jin, A. Bhat, D. Schlesinger, S. Son, D. A. Stevens, D. G. Drubin, D. A. Fletcher

This study shows that two FAST proteins use distinct actin nucleation pathways to drive cell-cell fusion, but their cytoplasmic tails are modular and functionally interchangeable.

bioRxiv · Jun 3, 2020Cell Biology

Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of cerebellar CHIPopathy

C.-h. Shi, C. Rubel, S. E. Soss, R. Sanchez-Hodge, S. Zhang, D. A. Stevens, H. McDonough, R. C. Page, W. Chazin, C. Patterson, C.-y. Mao, M. S. Willis, H.-Y. Luo, M.-b. Tang, P. Du, , J. Schisler

This study investigates the effects of a CHIP mutation in preclinical models, showing it causes structural disruption and functional loss linked to motor and cognitive deficits.

bioRxiv · Mar 15, 2018Animal Behavior & Cognition