Author

D. C. Bulmer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:electrophysiology· 1

Frequent Co-Authors

E. S. J. Smith· 2 shared J. P. Higham· 2 shared K. H. Barker· 2 shared A. J. Brown· 1 shared F. Welsh· 1 shared I. P. Chessell· 1 shared J. Ghooraroo· 1 shared J. R. Hockley· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPhysiology

E. S. J. Smith

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2 papers

Recent·Most cited

PhysiologybioRxivPreprintSep 12, 2023

GPR35 inhibits TRPA1-mediated colonic afferent hypersensitivity through suppression of Substance P release

R. A. Gupta, J. P. Higham, P. Urriola-Munoz…·D. C. Bulmer

This paper identifies GPR35 as a target for non-opioid analgesics by showing it inhibits TRPA1-mediated colonic hypersensitivity through suppression of Substance P release.

DOI 10.1101/2023.09.12.557415

mouseexperimental assay

𝕏 PostBluesky

PhysiologybioRxivPreprintOct 26, 2022

Sensitisation of colonic nociceptors by IL-13 is dependent on JAK and p38 MAPK activity

K. H. Barker, J. P. Higham, L. A. Pattison…·D. C. Bulmer

This study shows IL-13 sensitizes colonic nociceptors via JAK and p38 MAPK signaling, offering therapeutic targets for visceral pain in gastrointestinal diseases.

DOI 10.1101/2022.10.26.511877

mouseelectrophysiology

𝕏 PostBluesky