Author

D. F. Read

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Approach:deep learning· 1 Research Focus:differential expression· 1

Frequent Co-Authors

C. Trapnell· 2 shared A. Cheong· 1 shared A. R. Osterburg· 1 shared A. Telesnitsky· 1 shared C. M. Shaver· 1 shared C. Williams· 1 shared C.-H. Chang· 1 shared E. Atindaana· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersPathology

C. Trapnell

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pulmonary osteoclast-like cells in silica induced pulmonary fibrosis

Y. Hasegawa, J. M. Franks, Y. Tanaka, Y. Uehara, D. F. Read, C. Williams, S. Srivatsan, L. B. Pitstick, N. M. Nikolaidis, C. M. Shaver, H. Wu, J. C. Gardner, A. R. Osterburg, J. J. Yu, E. J. Kopras, , F. X. McCormack

The paper investigates silica-induced pulmonary fibrosis in mice, finding that silica triggers osteoclast-like differentiation of macrophages and monocytes, contributing to lung injury and fibrosis.

bioRxiv · Feb 17, 2023Pathology

Airway epithelial interferon response to SARS-CoV-2 is inferior to rhinovirus and heterologous rhinovirus infection suppresses SARS-CoV-2 replication

E. R. Vanderwall, K. A. Barrow, L. M. Rich, D. F. Read, C. Trapnell, O. Oghenemega, S. F. Ziegler, T. S. Hallstrand, M. P. White, J. S. Debley

The paper compares the IFN response to SARS-CoV-2 and HRV-16 in airway epithelial cells and shows that HRV-16 induces a stronger IFN response and reduces SARS-CoV-2 replication.

bioRxiv · Nov 20, 2021Immunology

Stable integrant-specific differences in bimodal HIV-1 expression patterns revealed by high-throughput analysis

D. F. Read, E. Atindaana, K. Pyaram, F. Yang, S. Emery, A. Cheong, K. R. Nakama, E. Larragoite, E. Battivelli, E. Verdin, V. Planelles, C.-H. Chang, A. Telesnitsky, J. M. Kidd

The paper tracks HIV-1 proviral clones over time in polyclonal populations, revealing stable, clone-specific expression patterns and clonal expansion dynamics independent of cell-type or metabolic factors.

PLOS Pathogens · Oct 4, 2019Peer-reviewedMicrobiology