Author

S. Southekal

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:multiome· 1 Assay:proteomics· 1

Frequent Co-Authors

C. Guda· 2 shared C. Rao· 2 shared K. W. Fisher· 2 shared R. A. Svoboda· 2 shared R. E. Lewis· 2 shared A. R. Black· 1 shared D. Chatterjee· 1 shared D. E. Frodyma· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

C. Guda

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cell BiologybioRxivPreprintMay 16, 2022

Post-transcriptional control of SRSF9 promotes the epithelial-to-mesenchymal transition (EMT) in colorectal cancer cells

C. Rao, R. A. Svoboda, S. Southekal…·R. E. Lewis

The paper shows that KSR1-dependent signaling in CRC cells promotes SRSF9-mediated splicing of EPSTI1 mRNA, which drives EMT and metastasis.

DOI 10.1101/2022.05.16.492181

𝕏 PostBluesky

Cancer BiologybioRxivJan 18, 2021

KSR1-and ERK-dependent Translational Regulation of the Epithelial-to-Mesenchymal Transition

C. Rao, D. E. Frodyma, S. Southekal…·R. E. Lewis

This study shows KSR1 and ERK promote EMT by selectively translating EPSTI1, which facilitates the E-to N-cadherin switch and enhances migration and invasion in CRC cells.

DOI 10.7554/elife.66608

humanproteomics

𝕏 PostBluesky