Author

D. Hubmacher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

C. Baldock· 1 shared M. Singh· 1 shared N. Taye· 1 shared Z. Balic· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

4 top collaborators · 3 mutual co-author links

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1 shared paperCell Biology

C. Baldock

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2 papers

Recent·Most cited

Cell BiologybioRxivDec 6, 2022

Secreted ADAMTS-like 2 promotes myoblast differentiation by potentiating Wnt signaling

D. Hubmacher, N. Taye, C. Baldock, M. Singh

This study identifies ADAMTSL2 as a regulator of myogenesis by enhancing Wnt signaling, showing its role in promoting myoblast differentiation and muscle development.

DOI 10.1016/j.matbio.2023.05.003

mouseexperimental assay

𝕏 PostBluesky

Cell BiologybioRxivPreprintNov 23, 2022

Promoting translational readthrough to augment fibrillin-1 (FBN1) deposition in Marfan syndrome fibroblasts: A proof-of-concept study.

Z. Balic, D. Hubmacher

This study shows that translational readthrough drugs like ataluren and gentamicin improve FBN1 deposition in Marfan syndrome fibroblasts with PTC variants.

DOI 10.1101/2022.11.23.517642

humanproteomics

𝕏 PostBluesky