Author

D. Lenggenhager

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Gaspert· 1 shared A. Poeschel· 1 shared A. Vukolic· 1 shared A. Weber· 1 shared A.-L. Leblond· 1 shared B. Helmchen· 1 shared B. Muellhaupt· 1 shared C. Cortijo· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPathology

A. Gaspert

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Author profile →

2 papers

Recent·Most cited

A HEV ORF2 protein-mediated mechanism of hepatitis E associated kidney disease.

B. Helmchen, A.-L. Leblond, D. Lenggenhager, J. Jetzer, M. Ankavay, F. Helmchen, H. Yurtsever, R. Parrotta, M. E. Healy, A. Poeschel, E. Markkanen, N. Semmo, M. Ferrie, L. Cocquerel, H. Seeger, , A. Weber

This study identifies HEV ORF2 protein as a cause of glomerulonephritis in hepatitis E patients through immune complex deposition, without detectable viral particles or RNA.

bioRxiv · Sep 14, 2023 · PreprintPathology

SF3B1 promotes tumor malignancy through splicing-independent co-activation of HIF1α

P. Simmler, C. Cortijo, L. M. Koch, P. Galliker, M. Schaefer, S. Angori, H. A. Bolck, C. Mueller, A. Vukolic, P. Mirtschink, Y. Christinat, N. R. Davidson, K.-V. Lehmann, G. Pellegrini, C. Pauli, , G. Schwank

This study shows SF3B1 promotes tumor malignancy by co-activating HIF1α through a splicing-independent mechanism, enhancing hypoxia response element binding and target gene expression.

bioRxiv · Nov 10, 2020Cell Biology