Author

E. Gracey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Approach:biophysical modeling· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

D. Elewaut· 2 shared E. Dumas· 2 shared I. Josipovic· 2 shared L. Vereecke· 2 shared A. Ramming· 1 shared A. Thiran· 1 shared A. V. Perruccio· 1 shared C. Pastrello· 1 shared

Activity by Year

2023-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

D. Elewaut

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cell and Transcriptomic Diversity of Infrapatellar Fat Pad during Knee Osteoarthritis

H. Peters, P. Potla, J. S. Rockel, T. Tockovska, C. Pastrello, I. Jurisica, K. D. Santos, S. Vohra, S. Lively, K. Perry, N. Looby, S. H. Li, V. Chandran, K. Hueniken, P. Kaur, , M. Kapoor

This study uses multi-omic approaches to analyze cellular and transcriptomic diversity in infrapatellar fat pad during knee osteoarthritis, identifying fibroblasts as key contributors to disease-related differences.

bioRxiv · Apr 4, 2024Cell Biology

GDF15 mediates inflammation-associated bone loss through a brain-bone axis

R. Van der Cruyssen, J. Devan, I. Heggli, D. Burri, D. Gaublomme, I. Josipovic, E. Dumas, C. Vlieghe, M. G. Raimondo, P. Zakharov, P. Jacques, S. De Mits, Z. Lukasik, M. Vuylsteke, T. Renson, , D. Elewaut

This study identifies GDF15 as a mediator of inflammation-associated bone loss via a brain-bone axis, showing it induces bone loss through GFRAL receptor activation in the hindbrain.

bioRxiv · Nov 22, 2023 · PreprintImmunology

Sterile triggers drive joint inflammation in TNF and IL-1beta dependent mouse arthritis models

A. Thiran, I. Petta, G. Blancke, M. Thorp, G. Plankaert, M. Jans, V. Andries, K. Barbry, T. Hochepied, C. Vanhove, E. Gracey, E. Dumas, T. Manuelo, I. Josipovic, G. van Loo, , L. Vereecke

This study shows that joint inflammation in TNF and IL-1beta mouse arthritis models occurs independently of the microbiota, while TNF-driven gut inflammation depends on it.

bioRxiv · Jan 31, 2023Immunology