Author

E. Gremese

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Approach:deep learning· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

B. Tolusso· 2 shared M. Kurowska-Stolarska· 2 shared S. Alivernini· 2 shared A. Antonopoulos· 1 shared A. Dell· 1 shared A. Elmesmari· 1 shared A. Filer· 1 shared A. Khan· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersImmunology

B. Tolusso

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Author profile →

2 papers

Recent·Most cited

COVID-19 and Rheumatoid Arthritis share myeloid pathogenic and resolving pathways

L. MacDonald, T. D. Otto, A. Elmesmari, B. Tolusso, D. Somma, C. McSharry, E. Gremese, I. B. McInnes, S. Alivernini, M. Kurowska-Stolarska

This study identifies shared myeloid pathways between COVID-19 pneumonitis and RA synovitis, highlighting MerTK as a therapeutic target for both conditions.

bioRxiv · Jul 26, 2020 · PreprintImmunology

Loss of α2-6 sialylation promotes the transformation of synovial fibroblasts into a pro-inflammatory phenotype in Rheumatoid Arthritis.

Y. Wang, A. Khan, A. Antonopoulos, L. Bouche, C. D. Buckley, A. Filer, K. Raza, K. Ping-Li, B. Tolusso, E. Gremese, M. Kurowska-Stolarska, S. Alivernini, A. Dell, S. M. Haslam, M. A. Pineda

This study shows that reduced α2-6 sialylation in synovial fibroblasts contributes to a pro-inflammatory phenotype in RA by altering glycosylation pathways.

bioRxiv · Mar 8, 2020Immunology