Author

E. Marcassa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

O. Florey· 2 shared R. Beale· 2 shared R. Ulferts· 2 shared A. Buckmelter· 1 shared A. Daley· 1 shared A. F. Lopez-Clavijo· 1 shared A. Gajbhiye· 1 shared A. H. Lystad· 1 shared

Activity by Year

2020-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCell Biology

O. Florey

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3 papers

Recent·Most cited

Subtractive CRISPR screen identifies factors involved in non-canonical LC3 lipidation.

R. Ulferts, E. Marcassa, L. Timimi, L. C. Lee, A. Daley, B. Montaner, S. D. Turner, O. Florey, J. K. Baillie, R. Beale

The paper uses a subtractive CRISPR screen to identify factors involved in non-canonical LC3 lipidation, revealing distinct roles for ATG4D and the RALGAP complex in different contexts.

bioRxiv · Nov 18, 2020 · PreprintCell Biology

Non-canonical autophagy drives alternative ATG8 conjugation to phosphatidylserine

J. Durgan, A. H. Lystad, K. Sloan, S. R. Carlsson, M. I. Wilson, E. Marcassa, R. Ulferts, J. Webster, A. F. Lopez-Clavijo, M. J. Wakelam, R. Beale, A. Simonsen, D. Oxley, O. Florey

This study shows that during non-canonical autophagy, ATG8 proteins are conjugated to phosphatidylserine instead of phosphatidylethanolamine, revealing a distinct lipidation mechanism.

bioRxiv · May 14, 2020Cell Biology

A novel USP30 inhibitor recapitulates genetic loss of USP30 and sets the trigger for PINK1-PARKIN amplification of mitochondrial ubiquitylation.

E. Rusilowicz-Jones, J. Jardine, A. Kallinos, A. Pinto-Fernandez, F. Guenther, M. Giurrandino, F. G. Barone, K. McCarron, C. J. Burke, A. Murad, A. Martinez, E. Marcassa, M. Gersch, A. Buckmelter, K. Kayser-Bricker, , M. J. Clague

The paper identifies a USP30 inhibitor that mimics genetic loss of USP30, revealing its role in regulating mitochondrial ubiquitylation and mitophagy through TOM complex components.

bioRxiv · Apr 16, 2020Cell Biology