Author

E. Nuebel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

J. Rutter· 2 shared C. Argyriou· 1 shared C. U. Kidwell· 1 shared D. Shimura· 1 shared I. Wittig· 1 shared J. A. Berg· 1 shared J. A. Maschek· 1 shared J. E. Cox· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

J. Rutter

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Author profile →

2 papers

Recent·Most cited

Protective mitochondrial fission induced by stress responsive protein GJA1-20k

D. Shimura, E. Nuebel, R. Baum, S. Xiao, J. S. Warren, T. Hong, J. Rutter, R. M. Shaw

This study identifies GJA1-20k as a stress-responsive protein that induces non-canonical mitochondrial fission to protect against ischemia-reperfusion injury.

bioRxiv · May 5, 2021Cell Biology

Msp1/ATAD1 restores mitochondrial function in Zellweger Spectrum Disease

E. Nuebel, J. T. Morgan, S. Fogarty, J. M. Winter, S. Lettlova, J. A. Berg, Y.-C. Chen, C. U. Kidwell, J. A. Maschek, K. J. Clowers, C. Argyriou, L. Chen, I. Wittig, J. E. Cox, M. Roh-Johnson, , J. Rutter

The paper investigates peroxisomal dysfunction in Zellweger Spectrum Disease and shows that overexpressing ATAD1 restores mitochondrial function in patient-derived fibroblasts.

bioRxiv · Sep 19, 2020 · PreprintCell Biology