Author

E. Schmid

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2 Approach:wet-lab / biochemical assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. Gessner· 1 shared A. Haghikia· 1 shared A. M. Joffe· 1 shared C. A. Akdis· 1 shared C. P. Davis· 1 shared D. A. Fletcher· 1 shared D. Mauro· 1 shared D.-J. Lee· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperImmunology

A. Gessner

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Gut-specific H3R signaling orchestrates microglia-dependent resolution of peripheral inflammation

K. Duerholz, M. Linnerbauer, E. Schmid, H. Danzer, L. Loesslein, L. Amend, L. Ehnes, M. Frech, V. Azizov, F. Schaelter, A. Gessner, S. Lucas, T.-R. Lesker, R. V. Taudte, J. Hofmann, , M. M. Zaiss

The paper identifies gut microbiota-derived histamine as a factor enabling the CNS to suppress peripheral inflammation in RA by activating H3R in the intestine and modulating microglia.

bioRxiv · Jul 11, 2024 · PreprintImmunology

NOP16 is a histone mimetic that regulates Histone H3K27 methylation and gene repression

K. Takashima, D.-J. Lee, M. F. Trovero, M. H. Rothi, M. Mistry, Y. Zhang, Z. Li, C. P. Davis, Z. Li, J. Natale, E. Schmid, J. Al Haddad, G. B. Hoffmann, S. Dietmann, S. H. Sui, , E. L. Greer

This study identifies NOP16 as a histone mimic that regulates H3K27 methylation and gene repression, showing its role in breast cancer progression.

bioRxiv · Jun 13, 2023 · PreprintCancer Biology

Size-dependent segregation controls macrophage phagocytosis of antibody-opsonized targets

M. H. Bakalar, A. M. Joffe, E. Schmid, S. Son, M. Podolski, D. A. Fletcher

This study identifies an antigen height threshold that regulates macrophage phagocytosis of antibody-opsonized targets, showing that antigens taller than 10 nm impair phagocytosis by disrupting Fc receptor signaling.

bioRxiv · Jan 19, 2018Cell Biology