Author

M. Mistry

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Approach:genetic perturbation / CRISPR· 2 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. Adler· 1 shared C. A. Lewis· 1 shared C. B. Clish· 1 shared C. P. Davis· 1 shared D. S. Hitchcock· 1 shared D.-J. Lee· 1 shared E. L. Greer· 1 shared E. Schmid· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

A. Adler

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

NOP16 is a histone mimetic that regulates Histone H3K27 methylation and gene repression

K. Takashima, D.-J. Lee, M. F. Trovero, M. H. Rothi, M. Mistry, Y. Zhang, Z. Li, C. P. Davis, Z. Li, J. Natale, E. Schmid, J. Al Haddad, G. B. Hoffmann, S. Dietmann, S. H. Sui, , E. L. Greer

This study identifies NOP16 as a histone mimic that regulates H3K27 methylation and gene repression, showing its role in breast cancer progression.

bioRxiv · Jun 13, 2023 · PreprintCancer Biology

Mild impairment of mitochondrial function increases longevity and pathogen resistance through ATFS-1-driven activation of p38-regulated innate immunity

J. Campos, Z. Wu, P. D. Rudich, S. Soo, M. Mistry, J. Ferreira, K. Blackwell, J. Van Raamsdonk

This paper shows that mild mitochondrial dysfunction in C. elegans extends lifespan and enhances pathogen resistance via ATFS-1-driven activation of p38-regulated innate immunity.

bioRxiv · Mar 31, 2021Cell Biology

Adaptation of pancreatic cancer cells to nutrient deprivation is reversible and requires glutamine synthetase stabilization by mTORC1

P.-Y. Tsai, M.-S. Lee, U. Jadhav, I. Naqvi, S. Madha, A. Adler, M. Mistry, S. Naumenko, C. A. Lewis, D. S. Hitchcock, F. R. Roberts, P. DelNero, T. Hank, K. C. Honselmann, V. Morales Oyarvide, , N. Y. Kalaany

This study shows that pancreatic cancer cells can adapt to nutrient deprivation by stabilizing glutamine synthetase via mTORC1, and this adaptation is reversible.

bioRxiv · Feb 16, 2020Cancer Biology