Author

F. Bormann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:DNA methylation· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Di Domenico· 1 shared A. Jarosch· 1 shared A. Menne· 1 shared A. Perren· 1 shared C. Bamford· 1 shared C. Sers· 1 shared D. Albrecht· 1 shared D. Capper· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

A. Di Domenico

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Author profile →

3 papers

Recent·Most cited

DNA methylation reveals distinct cells of origin for pancreatic neuroendocrine carcinomas (PanNECs) and pancreatic neuroendocrine tumors (PanNETs)

T. Simon, P. Riemer, K. Detjen, A. Di Domenico, F. Bormann, A. Menne, S. Khouja, N. Monje, L. H. Childs, D. Lenze, U. Leser, A. Jarosch, F. Rossner, M. Morkel, N. Bluethgen, , C. Sers

The paper uses DNA methylation analysis to distinguish high-grade PanNECs from other PanNETs, revealing an exocrine cell of origin for PanNECs.

bioRxiv · Jun 12, 2020Cancer Biology

Epigenetic deregulation of lamina-associated domains in Hutchinson-Gilford Progeria Syndrome

F. Koehler, F. Bormann, G. Raddatz, J. Gutekunst, T. Musch, F. Lyko, M. Rodriguez-Paredes

This study identifies epigenetic deregulation of lamina-associated domains in HGPS patient fibroblasts, linking it to gene expression changes and chromatin alterations.

bioRxiv · Jan 22, 2019Cell Biology

Differential induction of interferon stimulated genes between type I and type III interferons is independent of interferon receptor abundance

K. Pervolaraki, S. Rastgou Talemi, D. Albrecht, F. Bormann, C. Bamford, J. Mendoza, K. C. Garcia, J. McLauchlan, T. Höfer, M. L. Stanifer, S. Boulant

This paper shows that type I and III interferons induce interferon stimulated genes with different kinetics, and these differences are intrinsic to the signaling pathways, not receptor abundance.

bioRxiv · Oct 19, 2018Immunology