Author

F. M. Feringa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

R. H. Medema· 3 shared A. G. Manjon· 2 shared B. van den Broek· 2 shared J. van den Berg· 2 shared L. Koob· 2 shared L. Krenning· 2 shared A. Friskes· 1 shared E. S. Koeleman· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCell Biology

R. H. Medema

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MND1 enables repair of two-ended DNA double-strand breaks

L. Koob, A. Friskes, L. van Bergen, F. M. Feringa, B. van den Broek, E. S. Koeleman, M. Schubert, V. A. Blomen, T. R. Brummelkamp, L. Krenning, R. H. Medema

This study shows MND1 facilitates repair of two-ended DNA double-strand breaks in somatic cells through homologous recombination, but not one-ended breaks, and its absence increases IR-induced toxicity.

bioRxiv · Aug 24, 2022 · PreprintCell Biology

DNA end-resection in highly accessible chromatin produces a toxic break

J. van den Berg, S. E. P. Joosten, Y. Kim, A. G. Manjon, L. Krenning, L. Koob, F. M. Feringa, R. Klompmaker, B. van den Broek, K. Jalink, W. Zwart, R. H. Medema

The authors withdrew their manuscript due to inconsistencies in data and questionable experimental results regarding DNA end-resection and chromatin accessibility.

bioRxiv · Aug 30, 2021 · PreprintCell Biology

A limited number of double-strand DNA breaks are sufficient to delay cell cycle progression.

J. van den Berg, A. G. Manjon, K. Kielbassa, F. M. Feringa, R. Freire, R. H. Medema

The paper shows that a single DNA double-strand break can trigger a checkpoint response that delays cell cycle progression, and its abrogation leads to micronucleated daughter cells.

bioRxiv · May 7, 2018Cell Biology