Author

F. M. Powrie

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

M. Attar· 2 shared S. N. Sansom· 2 shared A. Easton· 1 shared A. Geremia· 1 shared A. L. Corbin· 1 shared D. L. Berthold· 1 shared D. Santhananthan· 1 shared E. Collantes· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

M. Attar

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintFeb 5, 2021

IL-1-driven stromal-neutrophil interaction in deep ulcers defines a pathotype of therapy non-responsive inflammatory bowel disease

M. Friedrich, M. Pohin, M. A. Jackson…·F. M. Powrie

DOI 10.1101/2021.02.05.429804

humanbulk RNA-seq

𝕏 PostBluesky

ImmunologybioRxivPreprintApr 7, 2019

IRF5 promotes intestinal inflammation by guiding monocyte differentiation towards a pathogenic CD11c+ macrophage phenotype

A. L. Corbin, M. Gomez-Vazquez, T. E. Khoyratty…·I. A. Udalova

This study shows IRF5 promotes pathogenic CD11c+ macrophage differentiation from monocytes in the gut, worsening inflammation during colitis.

DOI 10.1101/601963

mousebulk RNA-seq

𝕏 PostBluesky