Author

F. Prin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. P. Piovesan· 1 shared A. Shah· 1 shared C. Boogerd· 1 shared D. Hodzic· 1 shared E. Battey· 1 shared E. J. Ward· 1 shared E. Marhuenda· 1 shared F. M. Marelli-Berg· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. P. Piovesan

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2 papers

Recent·Most cited

Placental inflammation leads to abnormal embryonic heart development

E. J. Ward, S. Bert, S. Fanti, N. P. Dufton, K. M. Malone, R. T. Maughan, F. Prin, L.-K. Volpato, A. P. Piovesan, M. Perretti, F. M. Marelli-Berg, S. Nadkarni

This study shows that placental inflammation caused by neutrophils leads to abnormal embryonic heart development and postnatal cardiac dysfunction, which can be mitigated by reducing placental inflammation.

bioRxiv · Mar 10, 2022Immunology

Lem2 is essential for cardiac development by maintaining nuclear integrity

J. Ross, N. Arcos-Villacis, E. Battey, C. Boogerd, E. Marhuenda, D. Hodzic, F. Prin, T. Mohun, N. Catibog, O. Tapia, L. Gerace, T. Iskratsch, A. Shah, M. Stroud

This study shows Lem2 is essential for cardiac development by maintaining nuclear integrity, as its absence leads to DNA damage, nuclear rupture, and apoptosis in mouse hearts.

bioRxiv · Feb 10, 2022Cell Biology