Author

F. Ruemmele

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:differential expression· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:flow cytometry· 1

Frequent Co-Authors

F. Rieux-Laucat· 2 shared N. Cerf-Bensussan· 2 shared A. Bidaud-Meynard· 1 shared A. Bourdais· 1 shared B. Begue· 1 shared B. Neven· 1 shared C. B. Brugnara· 1 shared C. Benoist· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

F. Rieux-Laucat

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

V0-ATPase downregulation induces MVID-like brush border defects independently of apical trafficking in the mammalian intestine

A. Bidaud-Meynard, O. Nicolle, A. Bourdais, M. Duclos, J. Saleh, F. Ruemmele, H. F. Farin, D. Delacour, D. Moshous, G. Michaux

This study shows that V0-ATPase depletion causes MVID-like microvillus atrophy in mouse intestinal organoids without affecting apical trafficking.

bioRxiv · Nov 4, 2022Cell Biology

Single cell analysis of FOXP3 deficiencies in humans and mice unmasks intrinsic and extrinsic CD4+ T cell perturbations

D. Zemmour, L.-M. Charbonnier, J. Leon, E. Six, S. Keles, M. Delville, S. Baris, J. Zuber, K. Chen, B. Neven, M. I. Garcia-Lloret, F. Ruemmele, C. B. Brugnara, N. Cerf-Bensussan, F. Rieux-Laucat, , C. Benoist

The paper analyzes FOXP3 deficiencies in humans and mice using single-cell RNA-seq and flow cytometry to uncover intrinsic and extrinsic CD4+ T cell perturbations in IPEX syndrome.

bioRxiv · Jul 6, 2020Immunology

Network Analysis Of Inflammatory Bowel Disease Reveals PTPN2 As New Monogenic Cause Of Intestinal Inflammation

M. Parlato, J. Pazmandi, Q. Niam, F. Charbit-Henrion, B. Begue, E. Martin, M. Thian, F. Muller, M. Maggioni, R. Duclaux-Loras, F. Rieux-Laucat, T. J. Molina, S. Latour, F. Ruemmele, J. Menche, , N. Cerf-Bensussan

The paper uses network analysis to identify PTPN2 as a new monogenic cause of intestinal inflammation by integrating monogenic and polygenic IBD data.

bioRxiv · Sep 16, 2019 · PreprintImmunology