Author

F. Tafesse

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:metabolomics· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

C. Schultz· 2 shared S. E. Farley· 2 shared A. H. Worley· 1 shared A. Nellore· 1 shared D. M. Lewinsohn· 1 shared E. Karamooz· 1 shared E. W. Meermeier· 1 shared F. Stein· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersBiochemistry

C. Schultz

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Author profile →

3 papers

Recent·Most cited

Trifunctional fatty acid derivatives demonstrate the impact of diazirine placement

S. E. Farley, R. Hashimoto, J. Evangelista, F. Stein, P. Haberkant, K. Kikuchi, F. Tafesse, C. Schultz

The paper synthesizes three palmitic acid derivatives with diazirine at different positions and shows that end-position diazirines crosslink distinct protein subsets.

bioRxiv · May 15, 2024 · PreprintBiochemistry

A global lipid map reveals host dependency factors conserved across SARS-CoV-2 variants

S. E. Farley, J. Kyle, H. Leier, L. M. Bramer, J. Weinstein, T. A. Bates, J.-Y. Lee, T. O. Metz, C. Schultz, F. Tafesse

The paper identifies glycerolipid biosynthesis as a conserved host dependency factor for SARS-CoV-2 by mapping lipid metabolism changes across variants.

bioRxiv · Feb 14, 2022Microbiology

Alternative Splicing of MR1 regulates antigen presentation to MAIT cells

G. A. Narayanan, A. Nellore, J. Tran, A. H. Worley, E. W. Meermeier, E. Karamooz, M. Huber, R. Kurapova, F. Tafesse, M. J. Harriff, D. M. Lewinsohn

This study investigates how alternative splicing of MR1 affects antigen presentation to MAIT cells, finding that only MR1A can activate MAIT cells while MR1B inhibits their activation.

bioRxiv · Jul 11, 2019Immunology