Author

G. Apps

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1 Approach:genetic perturbation / CRISPR· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

A. Swarbrick· 1 shared C. C. Goodnow· 1 shared C. J. Jara· 1 shared C. Ma· 1 shared D. Suan· 1 shared E. Masle-Farquhar· 1 shared G. Al-Eryani· 1 shared G. Rao· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

A. Swarbrick

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

STAT3 gain-of-function mutations connect leukemia with autoimmune disease by pathological dysregulation of NKG2Dhi CD8 killer T cells

E. Masle-Farquhar, K. J. L. Jackson, T. J. Peters, G. Al-Eryani, M. Singh, K. J. Payne, G. Rao, G. Apps, J. Kingham, C. J. Jara, K. Skvortsova, A. Swarbrick, C. Ma, D. Suan, G. Uzel, , C. C. Goodnow

This study shows that STAT3 gain-of-function mutations in leukemia drive autoimmune pathology by causing NKG2Dhi CD8 T cell oligoclonal expansion and cytotoxic activity.

bioRxiv · Feb 11, 2022 · PreprintImmunology