Author

G. Pathare

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Salas-Bastos· 2 shared J. Loffing· 2 shared L. Sommer· 2 shared A. Francois· 1 shared C. A. Wagner· 1 shared C. Bardet· 1 shared C. Stockmann· 1 shared E. J. Yoo· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersPhysiology

A. Salas-Bastos

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Author profile →

2 papers

Recent·Most cited

Chronically elevated FGF23 drives sustained renal ERK signaling and inflammatory transcriptional programs mitigated by cFGF23 gene therapy

A. Salas-Bastos, C. Bardet, K. Kopper, L. Jauze, F. Collaud, A. Francois, G. Chen, M. Mohammadi, C. Stockmann, L. Sommer, J. Loffing, G. Ronzitti, G. Pathare

This study shows that chronic FGF23 elevation causes kidney inflammation via sustained ERK signaling, which is reduced by cFGF23 gene therapy.

bioRxiv · Jan 18, 2024 · PreprintPhysiology

Extracellular sodium regulates fibroblast growth factor 23 (FGF23) formation.

Z. Radvanyi, E. J. Yoo, P. Kandasamy, A. Salas-Bastos, S. Monnerat, J. Refardt, M. Christ-Crain, H. Hayashi, Y. Kondo, J. Jantsch, I. Rubio-Aliaga, L. Sommer, C. A. Wagner, M. A. Hediger, H. M. Kwon, , G. Pathare

This study shows that extracellular sodium concentration directly regulates FGF23 synthesis in osteoblast-like cells and links this regulation to NFAT5 expression, with implications for disease states like hyponatremia.

bioRxiv · Jun 23, 2023Physiology