Author

J. Jantsch

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Akbal· 1 shared A. Bozec· 1 shared A. Luhrmann· 1 shared A. Preikschat· 1 shared A. Salas-Bastos· 1 shared A. Schroeder· 1 shared B. Bodendorfer· 1 shared C. A. Wagner· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperImmunology

A. Akbal

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cytosolic sodium accumulation is a common danger signal triggering endocytic dysfunction and NLRP3 inflammasome activation

M. S. J. Mangan, A. Akbal, S. Rivara, A. Schroeder, G. L. Horvath, P. Walch, J. Hegermann, R. Kaiser, F. Duthie, J. Selkrig, J. Jantsch, R. Gerhard, D. Wachten, E. Latz

The paper shows that cytosolic sodium accumulation triggers NLRP3 inflammasome activation in response to various danger signals in human macrophages.

bioRxiv · Mar 26, 2024 · PreprintImmunology

Extracellular sodium regulates fibroblast growth factor 23 (FGF23) formation.

Z. Radvanyi, E. J. Yoo, P. Kandasamy, A. Salas-Bastos, S. Monnerat, J. Refardt, M. Christ-Crain, H. Hayashi, Y. Kondo, J. Jantsch, I. Rubio-Aliaga, L. Sommer, C. A. Wagner, M. A. Hediger, H. M. Kwon, , G. Pathare

This study shows that extracellular sodium concentration directly regulates FGF23 synthesis in osteoblast-like cells and links this regulation to NFAT5 expression, with implications for disease states like hyponatremia.

bioRxiv · Jun 23, 2023Physiology

Macrophages inhibit Coxiella burnetii by the ACOD1-itaconate pathway for containment of Q fever

L. Kohl, N. A. A. Siddique, B. Bodendorfer, R. Berger, A. Preikschat, C. Daniel, M. Oelke, M. Mauermeir, K.-T. Yang, I. Hayek, M. Szperlinski, J. Schulze-Luehrmann, U. Schleicher, A. Bozec, G. Kroenke, , R. Lang

This study identifies the ACOD1-itaconate pathway in macrophages as critical for restricting Coxiella burnetii replication and proposes it as a therapeutic target for Q fever.

bioRxiv · May 10, 2022Immunology