Author

H. A. Badmos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 2 Organism:Drosophila· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

D. Bennett· 2 shared A. Campbell· 1 shared N. Cobbe· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

3 top collaborators · 3 mutual co-author links

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2 shared papersCell Biology

D. Bennett

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintAug 3, 2022

Ataxin-7 promotes Drosophila posterior follicle cell maturation by suppressing yorkie function

H. A. Badmos, D. Bennett

This study shows Ataxin-7 suppresses yorkie function in Drosophila posterior follicle cells, essential for epithelial maturation and body axis specification.

DOI 10.1101/2022.08.03.502610

Drosophilagenetic perturbation / CRISPR

𝕏 PostBluesky

Cell BiologybioRxivJul 2, 2020

Drosophila USP22/non-stop regulates the Hippo pathway to polarise the actin cytoskeleton during collective border cell migration

H. A. Badmos, N. Cobbe, A. Campbell, D. Bennett

The paper identifies non-stop/USP22's role in Hippo pathway regulation during Drosophila border cell migration, showing its impact on actin polarization and cluster behavior.

DOI 10.1083/jcb.202007005

Drosophilaexperimental assay

𝕏 PostBluesky