Author

J. Poudrier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Assay:flow cytometry· 1

Frequent Co-Authors

M. Roger· 3 shared C. Tremblay· 2 shared J.-P. Routy· 2 shared K. Doyon-Laliberte· 2 shared A. C. Labbe· 1 shared B. Trottier· 1 shared C. A. Blish· 1 shared C. Chartrand-Lefebvre· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersImmunology

M. Roger

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3 papers

Recent·Most cited

Subclinical Atherosclerosis is Associated with Discrepancies in BAFF and APRIL Levels and Altered Breg Potential of Precursor-like Marginal Zone B-Cells in HIV Treated Individuals

M. Naegele Aranguren, K. Doyon-Laliberte, M. El-Far, C. Chartrand-Lefebvre, J.-P. Routy, J.-G. Baril, B. Trottier, C. Tremblay, M. Durand, J. Poudrier, M. Roger

The paper investigates the association between BAFF and APRIL levels and subclinical atherosclerosis in HIV-treated individuals, finding elevated BAFF correlates with CVD risk factors while APRIL shows inverse correlations.

bioRxiv · Aug 26, 2022Immunology

Excess BAFF Alters NR4As Expression Levels and Breg Function of Human Precursor-like Marginal Zone B-cells in the Context of HIV-1 infection

K. Doyon-Laliberte, M. Aranguren, M. Byrns, J. Chagnon-Choquet, M. Paniconi, J.-P. Routy, C. Tremblay, M.-C. Quintal, N. Brassard, D. E. Kaufmann, J. Poudrier, M. Roger

This study shows that excess BAFF reduces Breg function and NR4A expression in MZp B-cells from HIV-infected individuals, which may impair immune surveillance.

bioRxiv · Aug 15, 2022Immunology

TIGIT is upregulated by HIV-1 infection and marks a highly functional adaptive and mature subset of natural killer cells

E. Vendrame, C. Seiler, T. Ranganath, N. Q. Zhao, R. Vergara, M. Alary, A. C. Labbe, F. Guedou, J. Poudrier, S. Holmes, M. Roger, C. A. Blish

This study shows that TIGIT is upregulated on NK cells in untreated HIV+ individuals and marks a functional, mature NK cell subset with enhanced responses to HIV-infected targets.

bioRxiv · Sep 10, 2019Immunology