Author

J. R. Liang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Organism:human· 3 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:genetic perturbation / CRISPR· 1 Assay:proteomics· 1

Frequent Co-Authors

E. Lingeman· 2 shared J. E. Corn· 2 shared S. Ahmed· 2 shared J. Corn· 1 shared J. Farnung· 1 shared J. Olzmann· 1 shared J. W. Bode· 1 shared K. A. Tolmachova· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersCell Biology

E. Lingeman

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Author profile →

3 papers

Recent·Most cited

BiochemistrybioRxivPreprintAug 2, 2022

Human ATG3 contains a non-canonical LIR motif crucial for its enzymatic activity in autophagy

J. Farnung, M. Muhar, J. R. Liang…·J. W. Bode

The paper identifies a non-canonical LIR motif in ATG3 essential for its enzymatic activity in autophagy, showing its role in LC3 lipidation and thioester formation.

DOI 10.1101/2022.08.02.502437

humanproteomics

𝕏 PostBluesky

Cell BiologybioRxivFeb 25, 2019

A genome-wide screen for ER autophagy highlights key roles of mitochondrial metabolism and ER- resident UFMylation

J. R. Liang, E. Lingeman, T. Luong…·J. E. Corn

The paper identifies 200 factors involved in human ER-phagy through CRISPRi screening and reveals mitochondrial metabolism and ER-resident UFMylation as key regulators.

DOI 10.1016/j.cell.2020.02.017

humangenetic perturbation / CRISPR

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Cell BiologybioRxivMar 3, 2018

Atlastins mediate selective autophagy of the endoplasmic reticulum

J. R. Liang, E. Lingeman, S. Ahmed, J. Corn

This study identifies Atlastins as key regulators of ER-phagy, showing they facilitate ER membrane remodeling for autophagosomal engulfment and linking their dysfunction to hereditary spastic paraplegia.

DOI 10.1083/jcb.201804185

humanexperimental assay

𝕏 PostBluesky