Author

K. Szade

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1

Frequent Co-Authors

G. S. Gulati· 2 shared I. L. Weissman· 2 shared M. Zukowska· 2 shared A. Budzynski· 1 shared A. Jozkowicz· 1 shared A. Kusienicka· 1 shared A. Szade· 1 shared A. Zhang· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersCell Biology

G. S. Gulati

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Author profile →

3 papers

Recent·Most cited

Diosmetin alleviates liver inflammation by improving liver sinusoidal endothelial cell dysfunction.

D. Zurawek, N. Pydyn, P. Major, K. Szade, K. Trzos, E. Kus, E. Pospiech, P. Malczak, D. Radkowiak, A. Budzynski, S. Chlopicki, J. Jura, J. Kotlinowski

This paper shows diosmetin reduces liver inflammation by improving liver sinusoidal endothelial cell dysfunction in a mouse model.

Biomedicine & Pharmacotherapy · Feb 1, 2025Peer-reviewedCell Biology

Neogenin-1 distinguishes between myeloid-biased and balanced Hoxb5+ mouse long-term hematopoietic stem cells

G. S. Gulati, M. Zukowska, J. Noh, A. Zhang, R. Sinha, B. George, D. Wesche, I. L. Weissman, K. Szade

This paper identifies Neogenin-1 as a marker distinguishing myeloid-biased from balanced Hoxb5+ long-term hematopoietic stem cells in mice, revealing their functional heterogeneity and hierarchical relationship.

bioRxiv · Jun 26, 2019Cell Biology

Heme oxygenase-1 deficiency affects bone marrow niche and triggers premature exhaustion of hematopoietic stem cells.

K. Szade, M. Zukowska, A. Szade, W. N. Nowak, M. Ciesla, K. Bukowska-Strakova, G. S. Gulati, N. Kachamakova-Trojanowska, A. Kusienicka, E. Einwallner, J. Kijowski, S. Czauderna, H. Esterbauer, I. L. Weissman, J. Dulak, A. Jozkowicz

This study shows that HO-1 deficiency in the bone marrow niche leads to premature HSC exhaustion and aging features, which can be reversed by transplanting HO-1-expressing HSCs.

bioRxiv · Dec 10, 2018Cell Biology