Author

A. Jozkowicz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

K. Bukowska-Strakova· 2 shared A. Ambicka· 1 shared A. Ameur· 1 shared A. Johansson· 1 shared A. Klich-Raczka· 1 shared A. Kusienicka· 1 shared A. Piotrowski· 1 shared A. Szade· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

K. Bukowska-Strakova

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Immune cells lacking Y chromosome have widespread dysregulation of autosomal genes

J. P. Dumanski, J. Halvardson, H. Davies, E. Rychlicka-Buniowska, J. Mattisson, B. Torabi Moghadam, N. Nagy, K. Weglarczyk, K. Bukowska-Strakova, M. Danielsson, P. Olszewski, A. Piotrowski, E. Oerton, A. Ambicka, M. Przewoznik, , L. A. Forsberg

The paper examines immune cells with mosaic loss of the Y chromosome, finding widespread dysregulation of autosomal genes and linking this to immune dysfunction and disease.

bioRxiv · Feb 16, 2020Immunology

Heme oxygenase-1 deficiency affects bone marrow niche and triggers premature exhaustion of hematopoietic stem cells.

K. Szade, M. Zukowska, A. Szade, W. N. Nowak, M. Ciesla, K. Bukowska-Strakova, G. S. Gulati, N. Kachamakova-Trojanowska, A. Kusienicka, E. Einwallner, J. Kijowski, S. Czauderna, H. Esterbauer, I. L. Weissman, J. Dulak, A. Jozkowicz

This study shows that HO-1 deficiency in the bone marrow niche leads to premature HSC exhaustion and aging features, which can be reversed by transplanting HO-1-expressing HSCs.

bioRxiv · Dec 10, 2018Cell Biology