Author

L. Rubinelli

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Approach:biophysical modeling· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Di Lorenzo· 3 shared A. Marino· 1 shared C. Konrad· 1 shared C.-H. Lee· 1 shared G. Manfredi· 1 shared J. A. Maschek· 1 shared J. E. Ip· 1 shared J. Kelly· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCancer Biology

A. Di Lorenzo

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

PET imaging of fibroblast activation protein alpha (FAP) detects incipient cardiotoxicity due to anthracycline chemotherapy

C.-H. Lee, O. Manzo, L. Rubinelli, S. E. Carrasco, S. Cho, T. M. Jeitner, J. W. Babich, A. Di Lorenzo, J. Kelly

This study shows that PET imaging with [68Ga]Ga-FAPI-04 detects early cardiotoxicity from anthracycline chemotherapy before echocardiography, by targeting fibroblast activation protein alpha (FAP).

bioRxiv · Sep 3, 2023 · PreprintCancer Biology

S1P controls endothelial sphingolipid homeostasis via ORMDL

L. Sasset, K. H. Chowdhury, O. L. Manzo, L. Rubinelli, C. Konrad, J. A. Maschek, G. Manfredi, W. L. Holland, A. Di Lorenzo

This study identifies S1P as the sphingolipid sensed by endothelial cells via S1PRs, revealing the S1PR/ORMDLs axis as a regulatory unit controlling sphingolipid homeostasis.

bioRxiv · Oct 22, 2021 · PreprintCell Biology

Pressure overload leads to coronary plaque formation, progression and myocardial events in ApoE-/- mice

A. Marino, Y. Zhang, L. Rubinelli, J. E. Ip, A. Di Lorenzo

This study shows that pressure overload in ApoE-/- mice leads to coronary plaque formation, progression, and myocardial events, mimicking human coronary artery disease.

bioRxiv · Feb 26, 2019Pathology