Author

M. Bartish

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Organism:human· 1

Frequent Co-Authors

A. Emond· 2 shared C. Goncalves· 2 shared D. Plourde· 2 shared E. Khoury· 2 shared F. Huang· 2 shared J. Su· 2 shared Q. Guo· 2 shared W. H. Miller· 2 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

A. Emond

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

VEGF-C expression in TAMs rewires the metastaticfate of breast cancer cells

K. S. Banjerjee, T. Bekkhus, T. Kerzel, S. d. S. Ferreira, T. Wallmann, M. Wallerius, L. S. Landwehr, D. A. Agardy, J. Bergh, M. Bartish, J. Hartman, A. Ostman, M. L. Squadrito, C. Rolny

This study shows that VEGF-C-expressing TAMs reduce lung metastasis while increasing lymph node metastasis by normalizing tumor blood vessels and promoting lymphangiogenesis.

bioRxiv · Jan 26, 2022 · PreprintCancer Biology

The pleiotropic effects of the MNK1/2-eIF4E axis support immune suppression and metastasis in a model of postpartum breast cancer.

Q. Guo, M. Bartish, C. Goncalves, F. Huang, S. S. Krisna, S. Preston, A. Emond, V. Z. Li, C. Duerr, Y. Gui, A. Cleret-Buhot, P. Thebault, H. Lefrere, L. Lenaerts, D. Plourde, , W. H. Miller

This study shows that the MNK1/2-eIF4E axis promotes immune suppression and metastasis in postpartum breast cancer by affecting both tumor and non-tumor cells.

bioRxiv · Sep 7, 2020 · PreprintCancer Biology

The MNK1/2-eIF4E axis contributes to phenotype switching, melanoma progression, and resistance to immunotherapy.

F. Huang, C. Goncalves, M. Bartish, J. Remy-Sarrazin, Q. Guo, A. Emond, W. Yang, D. Plourde, J. Su, M. G. Gimeno, Y. Zhan, M. Attias, A. Galan, T. rzymski, M. Mazan, , S. del Rincon

This study identifies the MNK1/2-eIF4E axis as a driver of melanoma phenotype switching, progression, and immunotherapy resistance, with potential therapeutic targeting.

bioRxiv · May 29, 2020Cancer Biology