Author

M. Hamblin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. Shah· 1 shared B. Li· 1 shared D. M. Monack· 1 shared G. Mereddy· 1 shared J. H. Yang· 1 shared J. J. Collins· 1 shared M. L. Coldren· 1 shared M. Shaikh· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperMicrobiology

A. Shah

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2 papers

Recent·Most cited

KatG catalase deficiency confers bedaquiline hyper-susceptibility to isoniazid resistant Mycobacterium tuberculosis

N. Ofori-Anyinam, M. Hamblin, M. L. Coldren, B. Li, G. Mereddy, M. Shaikh, A. Shah, N. Ranu, S. Lu, P. C. Blainey, S. Ma, J. J. Collins, J. H. Yang

This study identifies KatG catalase deficiency in INH-resistant M. tuberculosis as a factor contributing to hyper-susceptibility to bedaquiline.

bioRxiv · Oct 17, 2023Microbiology

Salmonella enterica serovar Typhi uses two type 3 secretion systems to replicate in human macrophages and to colonize humanized mice

M. Hamblin, R. Schade, R. Narasimhan, D. M. Monack

This study shows that S. Typhi uses both T3SS-1 and T3SS-2 to replicate in human macrophages and colonize humanized mice, with functional redundancy between the systems.

bioRxiv · Jun 6, 2023 · PreprintMicrobiology