Author

M. Horioka

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

M. A. Kazmi· 2 shared T. Huber· 2 shared T. P. Sakmar· 2 shared A. Furstenberg· 1 shared A. R. Moore· 1 shared C. A. Rico· 1 shared E. Ceraudo· 1 shared E. Lorenzen· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

M. A. Kazmi

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Author profile →

2 papers

Recent·Most cited

Cancer BiologybioRxivJun 6, 2019

Uveal Melanoma Oncogene CYSLTR2 Encodes a Constitutively Active GPCR Highly Biased Toward Gq Signaling

E. Ceraudo, M. Horioka, J. M. Mattheisen…·T. Huber

DOI 10.1074/jbc.ra120.015352

humanproteomics

𝕏 PostBluesky

BiophysicsbioRxivMar 16, 2019

High-Affinity Binding of Chemokine Analogs that Display Ligand Bias at the HIV-1 Co-receptor CCR5

C. A. Rico, Y. A. Berchiche, M. Horioka…·T. Huber

The paper develops a fluorescence assay to study high-affinity chemokine analogs that show ligand bias at CCR5, finding they bind with subnanomolar affinities and occupy a site not accessible to native chemokines.

DOI 10.1016/j.bpj.2019.07.043

humanproteomics

𝕏 PostBluesky