Author

M. Inouye

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:GWAS / association mapping· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Bakshi· 1 shared A. C. Calkin· 1 shared A. D. Swafford· 1 shared A. P. Nath· 1 shared A. S. Havulinna· 1 shared A. Salim· 1 shared A. Zhuang· 1 shared B. G. Drew· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperGenetics

A. Bakshi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Loss of the Long Non-coding RNA OIP5-AS1 Exacerbates Heart Failure in a Sex-Specific Manner

A. Zhuang, A. C. Calkin, S. Lau, H. Kiriazis, D. Donner, Y. Liu, S. Bond, S. Moody, E. Gould, T. Colgan, S. Ruiz-Carmona, M. Inouye, T. Q. de Aguiar Vallim, E. Tarling, G. Quaife-Ryan, , B. G. Drew

This study shows that loss of OIP5-AS1 worsens heart failure in female mice but not males, suggesting a sex-specific role in cardiac function.

bioRxiv · Jan 28, 2021Physiology

Bacterial modification of the host glycosaminoglycan heparan sulfate modulates SARS-CoV-2 infectivity

C. Martino, B. P. Kellman, D. R. Sandoval, T. M. Clausen, C. A. Marotz, S. J. Song, S. Wandro, L. S. Zaramela, R. A. Salido Benitez, Q. Zhu, E. Armingol, Y. Vazquez-Baeza, D. McDonald, J. T. Sorrentino, B. Taylor, , R. Knight

This study shows that commensal bacteria modify heparan sulfate, reducing SARS-CoV-2 spike protein binding and that this bacterial presence decreases with age, increasing COVID-19 susceptibility.

bioRxiv · Aug 17, 2020 · PreprintMicrobiology

Neonatal genetics of gene expression reveal the origins of autoimmune and allergic disease risk

Q. Q. Huang, H. H. F. Tang, S. M. Teo, S. C. Ritchie, A. P. Nath, M. Brozynska, A. Salim, A. Bakshi, B. J. Holt, D. Mok, C. C. Khor, P. D. Sly, P. G. Holt, K. E. Holt, M. Inouye

The paper identifies gene expression quantitative trait loci (eQTLs) in neonatal immune cells and links them to autoimmune and allergic disease risk through colocalisation and Mendelian randomisation.

bioRxiv · Jun 27, 2019 · PreprintGenetics