Author

M. J. Gaffrey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1 Plant Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

M. J. MacCoss· 2 shared P. S. Rabinovitch· 2 shared T. Zhang· 2 shared W.-J. Qian· 2 shared A. Marshall-Colon· 1 shared B. James· 1 shared B. McKinley· 1 shared C. C. White· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersCell Biology

M. J. MacCoss

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Author profile →

3 papers

Recent·Most cited

Plant BiologybioRxivPreprintApr 22, 2023

The stem cell-type transcriptome of bioenergy sorghum reveals the spatial regulation of secondary cell wall networks

J. Fu, B. McKinley, B. James…·A. Marshall-Colon

The paper identifies cell-type specific gene expression and regulatory networks in bioenergy sorghum stems to enable engineering for biofuel production.

DOI 10.1101/2023.04.22.537921

plant (other)bulk RNA-seq

𝕏 PostBluesky

Cell BiologybioRxivAug 6, 2021

Elamipretide (SS-31) Treatment Attenuates Age-Associated Post-Translational Modifications of Heart Proteins

J. A. Whitson, M. Martin-Perez, T. Zhang…·P. S. Rabinovitch

This study investigates how elamipretide affects age-related post-translational modifications in mouse heart proteins, finding that it reverses oxidative changes but not phosphorylation changes.

DOI 10.1007/s11357-021-00447-6

mouseproteomics

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PhysiologybioRxivJan 2, 2020

Late-life restoration of mitochondrial function reverses cardiac dysfunction in old mice

Y. A. Chiao, H. Zhang, M. Sweetwyne…·P. S. Rabinovitch

This study shows that treating old mice with SS-31 improves cardiac diastolic function by reducing mitochondrial oxidative stress and restoring redox balance.

DOI 10.7554/elife.55513

mouseproteomics

𝕏 PostBluesky