Author

M. Kauer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell-cell communication· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1

Frequent Co-Authors

F. Rifatbegovic· 2 shared H. Sorger· 2 shared I. M. Ambros· 2 shared P. F. Ambros· 2 shared R. Windhager· 2 shared S. Taschner-Mandl· 2 shared T. Weiss· 2 shared A. Bileck· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCell Biology

F. Rifatbegovic

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

YAP1 is a key regulator of EWS::FLI1-dependent malignant transformation upon IGF-1 mediated reprogramming of bone mesenchymal stem cells

H. Kovar, R. Noorizadeh, B. Sax, T. Javaheri, B. Radic Sarikas, V. Fock, M. Kauer, A. Bykov, V. Suresh, M. Schlederer, L. Kenner, G. Weber, W. Mikulits, F. Halbritter, R. Moriggl

The paper shows that IGF-1 reprograms bone mesenchymal stem cells in EWS::FLI1-mutant mice, leading to tumor formation, with Yap1 playing a central role, and suggests combined IGF-1R and YAP/TEAD inhibition as a potential treatment.

bioRxiv · Jul 15, 2024Cancer Biology

Human repair-related Schwann cells adopt functions of antigen-presenting cells in vitro

J. Berner, T. Weiss, H. Sorger, F. Rifatbegovic, M. Kauer, R. Windhager, A. Dohnal, P. F. Ambros, I. M. Ambros, P. Steinberger, S. Taschner-Mandl

This study shows human repair-related Schwann cells exhibit antigen-presenting cell features and suppress T-cell activation in vitro.

bioRxiv · Mar 7, 2022Cell Biology

Schwann cell plasticity regulates neuroblastic tumor cell differentiation via epidermal growth factor-like protein 8

T. Weiss, S. Taschner-Mandl, A. Bileck, F. Rifatbegovic, H. Sorger, M. Kauer, C. Frech, R. Windhager, C. Gerner, P. F. Ambros, I. M. Ambros

This study shows that Schwann cells in neuroblastic tumors and injured nerves share repair-related functions, with EGFL8 promoting neuronal differentiation of tumor cells.

Nature Communications · Mar 12, 2021Peer-reviewedCancer Biology