Author

M. Marras

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 2

Signature Assays, Organisms & Methods

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Assay:electrophysiology· 2 Approach:experimental assay· 2 Approach:biophysical modeling· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

C. Abella· 2 shared J. Cui· 2 shared J. R. Silva· 2 shared J. Shi· 2 shared A. Bedi· 1 shared D. Zinn· 1 shared G. Zhang· 1 shared H. Liang· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersBiophysics

C. Abella

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Arrhythmia-associated Calmodulin Variants Interact with KCNQ1 to Confer Aberrant Membrane Trafficking and Function

P. w. Kang, L. Woodbury, P. Angsutararux, N. Sambare, J. Shi, M. Marras, C. Abella, A. Bedi, D. Zinn, J. Cui, J. R. Silva

This study investigates how arrhythmia-associated calmodulin variants affect KCNQ1 binding, membrane trafficking, and function, revealing variable impacts on cardiac ion channel regulation.

bioRxiv · Jan 28, 2023Biophysics

An allosteric agonist activates BK channels by perturbing coupling between Ca2+ binding and pore opening

G. Zhang, X. Xu, Z. Jia, Y. Geng, H. Liang, J. Shi, M. Marras, C. Abella, K. L. Magleby, J. R. Silva, J. Chen, X. Zou, J. Cui

The paper identifies BC5, an allosteric agonist that modulates BK channel activation by disrupting the coupling between Ca2+ binding and pore opening.

bioRxiv · Jan 28, 2022 · PreprintBiophysics