Author

M. Pajic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:proteomics· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:drug response / drug discovery· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. Johns· 2 shared A. Ashworth· 1 shared A. C. James· 1 shared A. Gulati· 1 shared A. J. Gill· 1 shared A. Kaur· 1 shared A. Mawson· 1 shared A. Van Oosterum· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersCancer Biology

A. Johns

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Author profile →

3 papers

Recent·Most cited

βIII-Tubulin is a Brake on Extrinsic Cell-Death in Pancreatic Cancer

J. Kokkinos, G. Sharbeen, R. M. C. Ignacio, E. Pandzic, J. Youkhana, C. Boyer, K. S. Haghighi, M. Gunawarman, D. Goldstein, V. Gebski, M. Pajic, O. Pitiyarachchi, M. E. Davis, G. Schulstad, O. S. M. Arkell, , P. A. Phillips

This study identifies βIII-tubulin as a suppressor of extrinsic apoptosis in pancreatic cancer, showing that its silencing enhances sensitivity to death signals and reduces tumor progression.

bioRxiv · Sep 29, 2022 · PreprintCancer Biology

PGRMC1 phosphorylation and cell plasticity 1: glycolysis, mitochondria, tumor growth

B. M. Thejer, P. P. Adhikary, A. Kaur, S. L. Teakel, A. Van Oosterum, I. Seth, M. Pajic, K. M. Hannan, M. Pavy, P. Poh, J. A. Jazayeri, T. Zaw, D. Pascovici, M. Ludescher, M. Pawlak, , M. A. Cahill

This study shows that altering PGRMC1 phosphorylation in cancer cells affects energy metabolism, mitochondrial function, and tumor growth.

bioRxiv · Sep 6, 2019Cancer Biology

Targeting DNA damage response and Replication Stress in Pancreatic Cancer

S. Dreyer, R. Upstill-Goddard, V. Paulus-Hock, C. Paris, E. Maria-Lampraki, E. Dray, B. Serrels, S. Rebus, H. Brunton, C. Nourse, U.-M. Bailey, M. Jones, K. Moran-Jones, D. Wright, F. Duthie, , Lord

The paper identifies novel therapeutic strategies for pancreatic cancer by analyzing DNA damage response and replication stress in patient-derived cell lines.

bioRxiv · Jul 24, 2019Cancer Biology