Author

M. Persson

AstraZeneca (Sweden)

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Desrichard· 1 shared A. Ho· 1 shared A. Salamon· 1 shared Ana L Gomes dos Santos· 1 shared Annette BakiD· 1 shared B. Kull· 1 shared C. Antonescu· 1 shared Cristian Rivera· 1 shared

Activity by Year

2017-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCancer Biology

A. Desrichard

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Reversible peptide self-assembly enables sustained drug delivery with tuneable pharmacokinetics

Therese W. Herling, Jiapeng Wei, Sivaneswary Genapathy, Cristian Rivera, M. Persson, Peter Gennemark, David G. Workman, Dan Lundberg, Elise Bernard, Hannah L. Bolt, Marianna Yanez Arteta, Sarah Will, Annette Bak, David C. Hornigold, Tuomas P. J. Knowles, Ana L Gomes dos Santos

This paper demonstrates that reversible peptide self-assembly can be used to achieve tuneable pharmacokinetics for therapeutic peptides, as shown with pramlintide in a rat model.

bioRxiv · Mar 27, 2026 · PreprintBiophysics

Mouse Model of Heart Attack and Stroke Shows Improved Survival with MPO Inhibition

S. Shamsuzzaman, R. A. Deaton, H. Doviak, M. Evans, A. Salamon, S. Karnewar, V. Serbulea, G. F. Alencar, L. S. Shankman, K. Walsh, S. Bekiranov, O. Kocher, M. Kreiger, B. Kull, M. Persson, , G. K. Owens

The paper introduces a mouse model of advanced atherosclerosis that exhibits spontaneous plaque rupture leading to MI or stroke, and shows that MPO inhibition with AZM198 reduces mortality and plaque rupture incidence.

Circulation · Aug 27, 2024Peer-reviewedPhysiology

Multi-dimensional genomic analysis of myoepithelial carcinoma identifies prevalent oncogenic gene fusions

M. Dalin, N. Katabi, M. Persson, K.-W. Lee, V. Makarov, A. Desrichard, L. Walsh, L. West, Z. Nadeem, D. Ramaswami, J. Havel, F. Kuo, K. Chadalavada, G. Nanjangud, N. Riaz, , L. G. T. Morris

This study identifies prevalent oncogenic gene fusions in myoepithelial carcinoma through genomic analysis, highlighting TGFBR3-PLAG1 as a key fusion and potential therapeutic target.

bioRxiv · Jun 9, 2017Cancer Biology