Author

K. Walsh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell type annotation· 2 Research Focus:drug response / drug discovery· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Assay:clinical cohort / trial· 1

Frequent Co-Authors

A. Fodor· 1 shared A. Hawley· 1 shared A. Hoyt-Miggelbrink· 1 shared A. Nelson· 1 shared A. P. Bhatt· 1 shared A. Patel· 1 shared A. S. Gulati· 1 shared A. Salamon· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 10 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperMicrobiology

A. Fodor

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Mouse Model of Heart Attack and Stroke Shows Improved Survival with MPO Inhibition

S. Shamsuzzaman, R. A. Deaton, H. Doviak, M. Evans, A. Salamon, S. Karnewar, V. Serbulea, G. F. Alencar, L. S. Shankman, K. Walsh, S. Bekiranov, O. Kocher, M. Kreiger, B. Kull, M. Persson, , G. K. Owens

The paper introduces a mouse model of advanced atherosclerosis that exhibits spontaneous plaque rupture leading to MI or stroke, and shows that MPO inhibition with AZM198 reduces mortality and plaque rupture incidence.

Circulation · Aug 27, 2024Peer-reviewedPhysiology

Giardia Antagonizes Beneficial Functions of Indigenous and Therapeutic Intestinal Bacteria during Malnutrition

A. P. Bhatt, J. Arnold, M. Awoniyi, S. Sun, V. Feijoli, P. H. Quintela, K. Walsh, R. Ngobeni, B. Hansen, A. S. Gulati, I. Carroll, M. A. Azcarate-Peril, A. Fodor, J. Swann, L. Bartelt

This study shows Giardia reduces the beneficial effects of Lactobacillus in malnourished mice, altering microbial balance and increasing intestinal damage.

bioRxiv · Jan 22, 2024 · PreprintMicrobiology

Glioblastoma and other intracranial tumors elicit systemic sympathetic hyperactivity that limits immunotherapeutic responses

S. J. Lorrey, L. Wachsmuth, J. Finlay, C. Neff, K. Ayasoufi, J. Waibl Polania, A. Hoyt-Miggelbrink, M. Price, L. Rein, S. Dell, R. Reesman, X. Cui, A. Hawley, E. Lerner, D. Wilkinson, , P. E. Fecci

This study shows that intracranial tumors cause systemic sympathetic hyperactivity, impairing T cell function and limiting immunotherapy, and that beta-adrenergic blockade improves outcomes in mouse models and human patients.

bioRxiv · Nov 2, 2023 · PreprintImmunology

The Cell Surface Receptors Ror1/2 Control Cardiac Myofibroblast Differentiation

N. W. Chavkin, S. Sano, Y. Wang, K. Oshima, K. Horitani, M. Sano, S. MacLauchlan, A. Nelson, K. Setia, T. Vippa, Y. Watanabe, J. J. Saucerman, K. K. Hirschi, N. Gokce, K. Walsh

This study shows that Ror1/2 receptors are essential for cardiac myofibroblast differentiation and heart remodeling in response to pressure overload.

bioRxiv · Mar 2, 2021Cell Biology