Author

M. Pertea

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. Gihawi· 1 shared A. Goenka· 1 shared A. Xu· 1 shared B. Erdogdu· 1 shared B. Hu· 1 shared B. R. Haddad· 1 shared C. Alamillo-Ferrer· 1 shared C. I. Wells· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. Gihawi

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2 papers

Recent·Most cited

Major data analysis errors invalidate cancer microbiome findings

A. Gihawi, Y. Ge, J. Lu, D. Puiu, A. Xu, C. S. Cooper, D. S. Brewer, M. Pertea, S. Salzberg

The paper re-analyzes a cancer microbiome study and identifies two major data and methodological errors that invalidate its findings and those of subsequent studies.

bioRxiv · Jul 28, 2023Cancer Biology

Temozolomide-induced guanine mutations create exploitable vulnerabilities of guanine-rich DNA and RNA regions in drug resistant gliomas

D. M. Tiek, B. Erdogdu, R. Razaghi, L. Jin, N. Sadowski, C. Alamillo-Ferrer, J. R. Hogg, B. R. Haddad, D. H. Drewry, C. I. Wells, J. E. Pickett, X. Song, A. Goenka, B. Hu, S. L. Goldlust, , R. B. Riggins

This study identifies guanine mutations in TMZ-resistant gliomas that disrupt G4 structures and splice sites, creating vulnerabilities targeted by G4 stabilizers and splicing inhibitors.

bioRxiv · Nov 21, 2021Cancer Biology