Author

M. Ruiz-Ortega

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

C. A. Lindegaard· 1 shared C. Barbas· 1 shared C. Castillo· 1 shared C. Pyke· 1 shared D. R. Hansen· 1 shared D. Rodriguez-Puyol· 1 shared D. Serra· 1 shared H. C. Beck· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperPathology

C. A. Lindegaard

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

PathologybioRxivPreprintJan 16, 2023

Ccn2 deficiency causes smooth muscle cell de-differentiation and severe atherosclerosis in hyperlipidemic mice

J. H. Larsen, J. Stubbe, H. C. Beck…·L. B. Steffensen

DOI 10.1101/2023.01.16.524161

mousebulk RNA-seq

𝕏 PostBluesky

BiochemistrybioRxivFeb 18, 2020

Renal tubule Cpt1a overexpression protects from kidney fibrosis by restoring mitochondrial homeostasis

V. Miguel, J. Tituana, J. I. Herrero…·S. Lamas

This study shows that overexpressing Cpt1a in renal tubular epithelial cells reduces fibrosis by restoring mitochondrial function and bioenergetics in CKD models and human patients.

DOI 10.1172/jci140695

𝕏 PostBluesky