Author

S. Lamas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

C. Barbas· 2 shared J. I. Herrero· 2 shared J. Tituana· 2 shared R. Ramos· 2 shared V. Miguel· 2 shared A. Chaix· 1 shared A. Meseguer· 1 shared A. Olsson· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersPhysiology

C. Barbas

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cross regulation between the molecular clock and kidney inflammatory, metabolic and fibrotic responses

C. Rey-Serra, J. Tituana, T. Lin, J. I. Herrero, V. Miguel, C. Barbas, A. Meseguer, R. Ramos, D. Firsov, A. Chaix, S. Panda, S. Lamas

This study investigates the interaction between the molecular clock and kidney fibrosis in experimental models, finding that clock components influence inflammation and fibrosis, while fibrosis alters clock gene expression.

bioRxiv · May 18, 2022 · PreprintPhysiology

Causes of death in newborn C57BL/6J mice

S. C. Peneda, G. M. Morello, S. Lamas, A. Olsson, C. Gilbert

This study investigates causes of death in newborn C57BL/6J mice, finding that starvation was the most common cause, followed by stillbirth.

bioRxiv · Feb 25, 2020 · PreprintPathology

Renal tubule Cpt1a overexpression protects from kidney fibrosis by restoring mitochondrial homeostasis

V. Miguel, J. Tituana, J. I. Herrero, L. Herrero, D. Serra, P. Cuevas, C. Barbas, D. Rodriguez-Puyol, L. Marquez-Exposito, M. Ruiz-Ortega, C. Castillo, X. Sheng, K. Susztak, M. Ruiz-Canela, J. Salas-Salvado, , S. Lamas

This study shows that overexpressing Cpt1a in renal tubular epithelial cells reduces fibrosis by restoring mitochondrial function and bioenergetics in CKD models and human patients.

bioRxiv · Feb 18, 2020Biochemistry