Author

N. Papazian

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Assay:bulk RNA-seq· 2 Assay:flow cytometry· 2 Organism:human· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

T. Cupedo· 4 shared A. Broijl· 3 shared C. Fokkema· 3 shared P. Sonneveld· 3 shared R. Hoogenboezem· 3 shared S. Tahri· 3 shared F. Gay· 2 shared G. Van Beek· 2 shared

Activity by Year

2019-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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4 shared papersImmunology

T. Cupedo

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

ImmunologybioRxivPreprintJul 5, 2023

Single-cell transcriptomic analysis of NK cell dynamics in myeloma patients reveal persistent reduction of cytotoxic NK cells from diagnosis to relapse

S. Tahri, M. M. E. de Jong, C. Fokkema…·T. Cupedo

DOI 10.1101/2023.07.05.547295

𝕏 PostBluesky

ImmunologybioRxivPreprintMar 3, 2023

An IL-1β driven neutrophil-stromal cell axis fosters a BAFF-rich microenvironment in multiple myeloma

M. M. de Jong, C. Fokkema, N. Papazian…·T. Cupedo

The paper identifies a neutrophil-stromal cell feedback loop in multiple myeloma that promotes BAFF-rich inflammation, persisting after treatment and suggesting dual targeting strategies.

DOI 10.1101/2023.03.03.530773

humanbulk RNA-seq

𝕏 PostBluesky

ImmunologybioRxivPreprintJan 18, 2023

Immune-mediated tumor control in the 5TGM1 transfer model of multiple myeloma

Z. Kellermayer, S. Tahri, M. M. E. De Jong…·T. Cupedo

This study uses a murine model to show that C57Bl/6 mice can control multiple myeloma through IFN-gamma-dependent immune responses, highlighting the model's utility for studying anti-myeloma immunity.

DOI 10.1101/2023.01.18.524505

mouseflow cytometry

𝕏 PostBluesky

ImmunologybioRxivSep 16, 2019

Yap1-driven intestinal repair is controlled by group 3 innate lymphoid cells

M. Romera-Hernandez, P. Aparicio-Domingo, N. Papazian…·T. Cupedo

This study identifies that ILC3-driven intestinal repair relies on Hippo-Yap1 signaling and Src family kinases, independent of IL-22 and Stat3.

DOI 10.1016/j.celrep.2019.11.115

mousebulk RNA-seq

𝕏 PostBluesky