Author

P. Ceppi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 5

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Assay:bulk RNA-seq· 5 Organism:human· 4 Research Focus:cancer & tumor microenvironment· 3 Approach:experimental assay· 3 Research Focus:differential expression· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

P. N. Gollavilli· 4 shared V. Ramesh· 4 shared A. Schwab· 3 shared L. Pinna· 3 shared M. A. Siddiqui· 3 shared B. Parma· 2 shared I. A. Asangani· 2 shared M. E. Vazakidou· 2 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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4 shared papersCancer Biology

P. N. Gollavilli

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5 papers

Recent·Most cited

Pan-cancer analysis of pyrimidine metabolism reveals signaling pathways connections with chemoresistance role

V. Ramesh, M. Demirdizen, L. Pinna, T. K. Doktor, M. A. Siddiqui, P. Ceppi

The paper performs a pan-cancer analysis of pyrimidine metabolism, identifying pathway interactions linked to chemoresistance and showing that brequinar treatment reverses doxorubicin resistance.

bioRxiv · Dec 6, 2023Cancer Biology

Propionate reinforces epithelial identity and reduces aggressiveness of non-small cell lung carcinoma via chromatin remodelling

V. Ramesh, P. N. Gollavilli, L. Pinna, M. A. Siddiqui, S. T. Jakobsen, E. L. Boiteux, K. E. Olsen, O. N. Jensen, R. Siersbaek, K. Somyajit, P. Ceppi

This study identifies propionate's role in reducing NSCLC aggressiveness by promoting epithelial identity through chromatin remodeling.

bioRxiv · Jan 19, 2023Cancer Biology

Metabolic breakdown of non-small cell lung cancers by mitochondrial HSPD1 targeting

B. Parma, V. Ramesh, P. N. Gollavilli, A. M. Siddiqui, L. Pinna, A. Schwab, S. Marshall, S. Zhang, C. Pilarsky, F. Napoli, M. Volante, S. Urbanczyk, D. Mielenz, H. D. Schroeder, M. Stemmler, , P. Ceppi

This study identifies HSPD1 as a therapeutic target in NSCLC, showing its disruption leads to metabolic breakdown and suppressed tumor growth.

bioRxiv · Mar 9, 2021Cancer Biology

Thymidylate synthase drives the phenotypes of epithelial-to-mesenchymal transition in non-small cell lung cancer

M. A. Siddiqui, P. N. Gollavilli, V. Ramesh, B. Parma, A. Schwab, M. E. Vazakidou, R. Natesan, O. Saatci, I. Rapa, P. Bironzo, H. Schuhwerk, I. A. Asangani, O. Sahin, M. Volante, P. Ceppi

This study identifies thymidylate synthase (TS) as a driver of epithelial-to-mesenchymal transition (EMT) in non-small cell lung cancer (NSCLC), linking it to metastasis, chemotherapy resistance, and survival.

bioRxiv · Aug 13, 2020Cancer Biology

Thymidylate synthase maintains the de-differentiated state of aggressive breast cancers

A. Siddiqui, P. N. Gollavilli, A. Schwab, M. E. Vazakidou, P. Ersan, M. Ramakrishnan, D. Pluim, S. A. Coggins, O. Saatci, L. Annaratone, J. H. Schellens, B. Kim, I. A. Asangani, S. A. K. Rasheed, C. Marchio, , P. Ceppi

This study shows that thymidylate synthase promotes de-differentiation in aggressive breast cancers by supporting pyrimidine catabolism and EMT, correlating with poor prognosis.

Cell Death & Differentiation · Nov 1, 2019Peer-reviewedCancer Biology